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	<title>ASN &#187; Vertigo</title>
	<atom:link href="http://www.neurootology.org/keywords/vertigo/feed" rel="self" type="application/rss+xml" />
	<link>http://www.neurootology.org</link>
	<description>Archives for Sensology and Neurootology in Science and Practice</description>
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		<title>Hygroma. Report, a Case</title>
		<link>http://www.neurootology.org/archives/561</link>
		<comments>http://www.neurootology.org/archives/561#comments</comments>
		<pubDate>Wed, 09 Jun 2010 15:31:22 +0000</pubDate>
		<dc:creator>julia</dc:creator>
				<category><![CDATA[Diseases]]></category>
		<category><![CDATA[Neurology]]></category>
		<category><![CDATA[Hearing loss]]></category>
		<category><![CDATA[Hygroma]]></category>
		<category><![CDATA[Tinnitus]]></category>
		<category><![CDATA[Vertigo]]></category>

		<guid isPermaLink="false">http://www.neurootology.org/?p=561</guid>
		<description><![CDATA[Cystic hygroma was first described in 1828 and referred to as a “moist tumor.Today, this benign developmental tumor is recognized as a lymphatic malformation (also known as a lymphangioma).Cystic hygroma is a benign lymphatic malformation representing 5% to 6% of all benign tumors and 1% of all tumors (benign and malignant). Most (90%) cystic hygromas [...]]]></description>
			<content:encoded><![CDATA[<p>Cystic hygroma was first described in 1828 and referred to as a “moist tumor.Today, this benign developmental tumor is recognized as a lymphatic malformation (also known as a lymphangioma).Cystic hygroma is a benign lymphatic malformation representing 5% to 6% of all benign tumors and 1% of all tumors (benign and malignant). Most (90%) cystic hygromas will manifest by age two, although there are reported cases in the literature of cystic hygroma presenting in adults.Cystic hygromas are among the most common congenital neck masses. The symptoms initial can be asymptomatic, painless lesion unless infected or bleeding progression: Continues to increases in size, 90% symptomatic by age 2 years. They have a predilection for the left posterior triangle and manifest early in life. Although various locations of cystic hygromas are found in the literature, distribution: Neck (60%), other locations : Chest Wall, Mediastinum, Axilla; Rare locations: Inguinal región and retroperitoneal region.. Pathophysiology the Congenital lymphatic malformation can be a Lymphatic vessel fails to connect and drain. Surgical excision is the treatment of choice, but injection of a sclerosing agent into the cyst is an alternative procedure Pregnancies complicated by fetal cystic hygroma in the second and third trimesters are often associated with hydrops fetalis, oligohydramnios or intrauterine fetal death which may make genetic assessment more difficult. The results in large multiloculated cyst sometimes are associated conditions: Turner&#8217;s Syndrome, Noonan&#8217;s Syndrome, Down Syndrome.1-4<br />
The diagnosis must be early, a Hygroma is key to preventing its serious consequences. Unfortunately, early detection of this is sometimes difficult, because the symptoms may be subtle and may not appear in the beginning stages of growth. Also, headache, hearing loss, dizziness, and tinnitus are symptoms of any inner ear problems. Therefore, once the symptoms appear, a thorough ear examination and hearing test are essential the study the vestibular function through of vestibuloespinal, retinoocular, vestibuloocular systems in the Cranio-Corpo-Graphy (CCG), the Test of Balance(TOB) and the Computarized Electronystagmography (CNG).  in order to determine and orientate a better vestibular diagnosis.Computerized Tomography(CT) and Magnetic Resonance Imaging (MRI) are helpful in determining the location and size of a hygroma and also in planning its removal.<a href='http://www.neurootology.org/media/2010/06/Hygroma-Report-a-case.pdf'>Hygroma Report a case</a></p>
]]></content:encoded>
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		</item>
		<item>
		<title>Vestibular disorders in old age vertigo</title>
		<link>http://www.neurootology.org/archives/509</link>
		<comments>http://www.neurootology.org/archives/509#comments</comments>
		<pubDate>Tue, 05 Aug 2008 16:58:29 +0000</pubDate>
		<dc:creator>julia</dc:creator>
				<category><![CDATA[Diseases]]></category>
		<category><![CDATA[Equilibrium]]></category>
		<category><![CDATA[Neurootology]]></category>
		<category><![CDATA[old age vertigo]]></category>
		<category><![CDATA[presbyataxia]]></category>
		<category><![CDATA[presbyvertigo]]></category>
		<category><![CDATA[Vertigo]]></category>

		<guid isPermaLink="false">http://www.neurootology.org/?p=509</guid>
		<description><![CDATA[Good health is not only a privilege of young generation but also of elderly people. Nowadays 13% of the population has reached the age of 65, and one third of them have vertiginous attack at least once in their life. 45-70% of the elderly has one or more drop attacks yearly. In the Semmelweis University [...]]]></description>
			<content:encoded><![CDATA[<p><!--StartFragment--></p>
<p class="MsoNormal"><span lang="EN-US">Good health is not only a privilege of young generation but also of elderly people. Nowadays 13% of the population has reached the age of 65, and one third of them have vertiginous attack at least once in their life. 45-70% of the elderly has one or more drop attacks yearly<strong>.</strong></span><span lang="EN-US"> </span></p>
<p class="MsoNormal"><span lang="EN-US">In the Semmelweis University Otoneurological Department, 17% of the patients are more than 65 years old. Detailed audiological and otoneurological examination was performed.</span></p>
<p class="MsoNormal"><span lang="EN-US">The origin of vertigo was extravestibular, like depression and anxiety disorder in 28% of the patients and vascular in 42%. Only in 30% specific balance system disease was observed.</span></p>
<p class="MsoNormal"><span lang="EN-US">Together with presbyacousia a slight loss in balancing appears. Gradually, elderly patients start noticing some unsteadiness or fear to keep moving. The presbyvertigo and presbyataxia are caused by the slow degeneration of vestibular end organs and brainstem pathways. The deterioration of sensorial organs and the central signal processing problems mean that our brain is getting old due to age. </span></p>
<p class="MsoNormal"><span lang="EN-US">The reasons of the old age vertigo are the decreased physical activity, concomitant diseases, increasing occurrence of vascular risk factors, degeneration of the cervical mechanoreceptors, and anxiety disorders with depression. The possibilities of the treatment: vasoactive and neuroprotective drug administration, adequate treatment of the concomitant diseases. The early mobilization of patients with vertigo is very important so as to avoid sedative drug administration.<span>  </span></span></p>
<p class="MsoNormal"><span lang="EN-US">By means of improving diagnostic procedures, successful prevention and therapy the disability can be avoided.</span></p>
<p><!--EndFragment--></p>
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		</item>
		<item>
		<title>Neurootological findings in patients, suffering from occipital skull fractures</title>
		<link>http://www.neurootology.org/archives/481</link>
		<comments>http://www.neurootology.org/archives/481#comments</comments>
		<pubDate>Thu, 03 Jul 2008 17:46:15 +0000</pubDate>
		<dc:creator>julia</dc:creator>
				<category><![CDATA[Uncategorized]]></category>
		<category><![CDATA[Dizziness]]></category>
		<category><![CDATA[hering loss]]></category>
		<category><![CDATA[skull fractures]]></category>
		<category><![CDATA[Vertigo]]></category>
		<category><![CDATA[Whiplash Injury]]></category>

		<guid isPermaLink="false">http://www.neurootology.org/?p=481</guid>
		<description><![CDATA[Skull traumas play an important roll in neurootological expertise writing. Skull traumas may occur at the rhinobasis, i.e. frontally, at the otobasis, i.e. laterally as well as from behind, i.e. occipitally. For this paper we have elected 26 cases of occipital skull fractures which were verified by radiology. It is known that the shockwaves of [...]]]></description>
			<content:encoded><![CDATA[<p>Skull traumas play an important roll in neurootological expertise writing. Skull traumas may occur at the rhinobasis, i.e. frontally, at the otobasis, i.e. laterally as well as from behind, i.e. occipitally. For this paper we have elected 26 cases of occipital skull fractures which were verified by radiology. It is known that the shockwaves of an occipital skull fracture does not only lead to local tissue damages in the neighbouring occipital lobe and/or cerebellum. But pressure waves may induce contrecoup effects at the frontal lobe and at the brainstem.</p>
<p>Due to the brainstem involvement we find a higher rate of vertigo complaints and of vertigo releasing factors in our randomly selected sample of 26 cases. The nausea complaints are lagging behind the vertigo complaints. The vertigo complaints are long-lasting. There also is an elevated percentage of oculomotor regulating disturbances with double vision and oscillopsia. The elevated percentage of anosmia must be explained by a contrecoup effect. Approximately half of the sample is suffering from tinnitus and subjective hearing losses.</p>
<p>In equilibriometry  by means of polygraphic ENG we found much elevation in spontaneous nystagmus in lying position more then in sitting position. By means of the trinary coded caloric Claussen butterfly we detected 55% of pathology. The trinary stepping CCG is not as sensitive as it only detected in 47,3% of pathology. The complaints about a subjective hearing loss are more intensively objectivated by speech audiometry than by pure tone audiometry.</p>
]]></content:encoded>
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		</item>
		<item>
		<title>Current trends of neurootological pharmacotherapy</title>
		<link>http://www.neurootology.org/archives/458</link>
		<comments>http://www.neurootology.org/archives/458#comments</comments>
		<pubDate>Mon, 23 Jun 2008 22:50:33 +0000</pubDate>
		<dc:creator>julia</dc:creator>
				<category><![CDATA[Neurootologic therapy]]></category>
		<category><![CDATA[Neurootology]]></category>
		<category><![CDATA[Pharmacotherapy]]></category>
		<category><![CDATA[Añadir nueva etiqueta]]></category>
		<category><![CDATA[Treatment]]></category>
		<category><![CDATA[Vertigo]]></category>

		<guid isPermaLink="false">http://www.neurootology.org/?p=458</guid>
		<description><![CDATA[Major neurootological complaints, which mostly need drug treatment, are: giddiness, dizziness, hearing loss and tinnitus. The neurootological differential diagnosis is the basis for planning the mostly supportive treatment of vertigo patients. In planning the therapy, we are utilizing a computerbased expert system CLAMEDEX for establishing the neurootological diagnosis through history, ORL inspection, ENG, calorics, rotatory [...]]]></description>
			<content:encoded><![CDATA[<p>Major neurootological complaints, which mostly need drug treatment, are: giddiness,</p>
<p>dizziness, hearing loss and tinnitus. The neurootological differential diagnosis is the basis</p>
<p>for planning the mostly supportive treatment of vertigo patients. In planning the therapy, we</p>
<p>are utilizing a computerbased expert system CLAMEDEX for establishing the</p>
<p>neurootological diagnosis through history, ORL inspection, ENG, calorics, rotatory chair test,</p>
<p>cranio-corpo-graphy (CCG), optokinetics, psychophysical audiometry, acoustic brainstem</p>
<p>and late evoked potentials, visually evoked potentials etc.. On this knowledge base we are</p>
<p>designing an individually adapted case oriented drug therapy.</p>
<p>Nausea and vomitus are the important subjective complications of dysequilibrium states.</p>
<p>Therefore antivertiginous and antiemetic therapies have to be applied if necessary. Usually</p>
<p>the duration is of limited time.</p>
<p>Oher drugs being chosen for a supportive pharmaco therapy according to the functional</p>
<p>topodiagnostics of the lesions usually possess one or more of the following actions upon the</p>
<p>equilibrium regulating network:</p>
<p>1. increase of cerebral blood flow,</p>
<p>2. enhanced penetration through the blood brain barrier,</p>
<p>3. increase of neuronal metabolism and</p>
<p>4. stabilization of specific neurotransmitters.</p>
<p>The neurootological patients are treated and monitored by regular neurootometric follow up</p>
<p>investigations.</p>
]]></content:encoded>
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		</item>
		<item>
		<title>Typical Caloric ENG Findings in the Claussen-Butterfly in Tinnitus Cases</title>
		<link>http://www.neurootology.org/archives/450</link>
		<comments>http://www.neurootology.org/archives/450#comments</comments>
		<pubDate>Mon, 23 Jun 2008 18:54:28 +0000</pubDate>
		<dc:creator>julia</dc:creator>
				<category><![CDATA[Diseases]]></category>
		<category><![CDATA[Hearing]]></category>
		<category><![CDATA[Neurootology]]></category>
		<category><![CDATA[caloric test]]></category>
		<category><![CDATA[Claussen-Butterfly]]></category>
		<category><![CDATA[Tinnitus]]></category>
		<category><![CDATA[Vertigo]]></category>

		<guid isPermaLink="false">http://www.neurootology.org/?p=450</guid>
		<description><![CDATA[The aim of this study was to investigate the different caloric findings in the Claussen-Butterflies in tinnitus patients under the assumption of a closely interrelated stato-acoustic network and to correlate these findings with our different tinnitus types. For this we used the NODEC IV data bank and searched for the most frequent types of butterflies [...]]]></description>
			<content:encoded><![CDATA[<p><!--StartFragment--></p>
<p class="MsoNormal"><span lang="EN-GB">The aim of this study was to investigate the different caloric findings in the Claussen-Butterflies in tinnitus patients under the assumption of a closely interrelated stato-acoustic network and to correlate these findings with our different tinnitus types.</span></p>
<p class="MsoNormal"><span lang="EN-GB">For this we used the NODEC IV data bank and searched for the most frequent types of butterflies in the tinnitus patients. From these patients we took a sample of 56, classified them into: endogenous tinnitus, exogenous tinnitus, combined tinnitus and bruit. Then we correlated each type with a closely related butterfly pattern. Only a minority suffered from objective and quantitative measurable peripheral vestibular lesions. However the majority revealed central vestibular lesions, more obvious together with endogenous tinnitus.</span></p>
<p><!--EndFragment--></p>
]]></content:encoded>
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		</item>
		<item>
		<title>Hamangioblastoma. Report a Case</title>
		<link>http://www.neurootology.org/archives/444</link>
		<comments>http://www.neurootology.org/archives/444#comments</comments>
		<pubDate>Fri, 06 Jun 2008 20:40:52 +0000</pubDate>
		<dc:creator>admin</dc:creator>
				<category><![CDATA[Diseases]]></category>
		<category><![CDATA[Equilibrium]]></category>
		<category><![CDATA[Neurootology]]></category>
		<category><![CDATA[hemangioblastoma]]></category>
		<category><![CDATA[Hipoacusia]]></category>
		<category><![CDATA[Tinnitus]]></category>
		<category><![CDATA[Vertigo]]></category>

		<guid isPermaLink="false">http://www.neurootology.org/?p=444</guid>
		<description><![CDATA[By Definition HEMANGIOBLASTOMA is a benign tumor that may develop on the nervous system that may occur sporadically or in association with Hippel-Lindau disease. It accounts for approximately 2% of intracranial tumors, arising most frequently in the cerebellar hemispheres and vermis. Histologically, the tumors are composed of multiple capillary and sinusoidal channels lined with endothelial [...]]]></description>
			<content:encoded><![CDATA[<p>By Definition HEMANGIOBLASTOMA is a benign tumor that may develop on the nervous system that may occur sporadically or in association with Hippel-Lindau disease. It accounts for approximately 2% of intracranial tumors, arising most frequently in the cerebellar hemispheres and vermis. Histologically, the tumors are composed of multiple capillary and sinusoidal channels lined with endothelial cells and clusters of lipid-laden pseudoxanthoma cells. Usually solitary, these tumors can be multiple and may also occur in the brain stem, spinal cord, retina, and supratentorial compartment. Cerebellar hemangioblastomas usually present in the third decade with intracranial hypertension, and ataxia. (From DeVita et al., Cancer: Principles and Practice of Oncology, 5th ed, pp2071-2)</p>
<p>We report  one patient with headache and symptoms of cerebellum involvement.</p>
<p>MRI showed a cerebellar large mass compatible with hemangioblastoma.</p>
<p>Often causing gradual hearing loss, tinnitus  and dizziness.If the tumor becomes large, it can interfere with the facial nerve, causing partial paralysis, and eventually pressing against brain structures, becoming life-threatening,</p>
<p>The diagnosis must be early, an hemangioblastoma is key to preventing its serious consequences. Unfortunately, early detection of the tumor is sometimes difficult, because the symptoms may be subtle and may not appear in the beginning stages of growth. Also, hearing loss, dizziness, and tinnitus are common symptoms of any middle and inner ear problems. Therefore, once the symptoms appear, a thorough ear examination and hearing test are essential the study the vestibular function through of vestibuloespinal, retinalocular, vestibuloocular systems in the Cranio-Corpo-Graphy (CCG), the Test of Balance(TOB) and the Computarized Electronystagmography (CNG).  in order to determine and orientate a better vestibular diagnosis. Computerized tomography(CT) and magnetic resonance imaging (MRI) are helpful in determining the location and size of a tumor and also in planning its removal.</p>
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		</item>
		<item>
		<title>Acustic neurinoma. report a case.</title>
		<link>http://www.neurootology.org/archives/291</link>
		<comments>http://www.neurootology.org/archives/291#comments</comments>
		<pubDate>Sun, 01 Jan 2006 00:00:00 +0000</pubDate>
		<dc:creator>admin</dc:creator>
				<category><![CDATA[Diseases]]></category>
		<category><![CDATA[Equilibrium]]></category>
		<category><![CDATA[Neurootologic therapy]]></category>
		<category><![CDATA[Neurootology]]></category>
		<category><![CDATA[Surgery]]></category>
		<category><![CDATA[Acustic nerinom]]></category>
		<category><![CDATA[Hipoacusia]]></category>
		<category><![CDATA[Tinnitus]]></category>
		<category><![CDATA[Vertigo]]></category>

		<guid isPermaLink="false">http://www.neurootology.org/?p=291</guid>
		<description><![CDATA[By Definition Acoustic Neurinoma is a benign tumor that may develop on the hearing and balance nerves near the inner ear. The tumor results from an overproduction of Schwann cells &#8211; small, sheet-like cells that normally wrap around nerve fibers like onion skin and help support the nerves. When growth is abnormally excessive, Schwann cells [...]]]></description>
			<content:encoded><![CDATA[<p>By Definition Acoustic Neurinoma is a benign tumor that may develop on the hearing and balance nerves near the inner ear. The tumor results from an overproduction of Schwann cells &#8211; small, sheet-like cells that normally wrap around nerve fibers like onion skin and help support the nerves. When growth is abnormally excessive, Schwann cells bunch together, pressing against the hearing and balance nerves, often causing gradual hearing loss, tinnitus  and dizziness. If the tumor becomes large, it can interfere with the facial nerve, causing partial paralysis, and eventually pressing against brain structures, becoming life-threatening. There are two types of acoustic neurinomas: Unilateral neurinomas account for approximately 5-8 percent of tumors inside the skull. Symptoms may develop at any age, but usually occur between the ages of 30 and 60 years. Bilateral acoustic neurinomas, affect both ears, are hereditary. Inherited from one&#8217;s parents, this tumor results from a genetic disorder known as neurofibromatosis-2 (NF2). Scientists believe that both types of acoustic neurinoma form following a loss of the function of a gene on chromosome 22.</p>
<p>The diagnosis must be early, an acoustic neurinoma is key to preventing its serious consequences. Unfortunately, early detection of the tumor is sometimes difficult, because the symptoms may be subtle and may not appear in the beginning stages of growth. Also, hearing loss, dizziness, and tinnitus are common symptoms of any middle and inner ear problems. Therefore, once the symptoms appear, a thorough ear examination and hearing test are essential the study the vestibular function through of vestibuloespinal, retinalocular, vestibuloocular systems in the Cranio-Corpo-Graphy (CCG), the Test of Balance(TOB) and the Computarized Electronystagmography (CNG). In order to determine and orientate a better vestibular diagnosis. Auditory Evoked Potentials, Computerized tomography(CT) and magnetic resonance imaging (MRI) are helpful in determining the location and size of a tumor and also in planning its removal.</p>
]]></content:encoded>
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		</item>
		<item>
		<title>Die moderne diagnostik bei gleichgewichtsfunktionsstörungen einschließlich eng, ccg und vbep</title>
		<link>http://www.neurootology.org/archives/292</link>
		<comments>http://www.neurootology.org/archives/292#comments</comments>
		<pubDate>Sun, 01 Jan 2006 00:00:00 +0000</pubDate>
		<dc:creator>admin</dc:creator>
				<category><![CDATA[Diseases]]></category>
		<category><![CDATA[Equilibriometric tests]]></category>
		<category><![CDATA[Equilibrium]]></category>
		<category><![CDATA[Neurootology]]></category>
		<category><![CDATA[Balance control]]></category>
		<category><![CDATA[Balance disorders]]></category>
		<category><![CDATA[Brain mapping]]></category>
		<category><![CDATA[Craniocorpografia digitale]]></category>
		<category><![CDATA[Craniocorpography]]></category>
		<category><![CDATA[Electronystagmography]]></category>
		<category><![CDATA[Equilibrium system]]></category>
		<category><![CDATA[Vertigo]]></category>
		<category><![CDATA[Vestibular evoked potentials]]></category>

		<guid isPermaLink="false">http://www.neurootology.org/?p=292</guid>
		<description><![CDATA[Die menschliche Gleichgewichtsfunktion wird durch ein sogenanntes Raumkonzept mit Einspeisung von Informationen aus der Gleichgewichtstetrade, nämlich Auge, Vestibularis, Cochlea und Propriozept im menschlichen Hirnstamm gebildet. Ergänzt wird die Gleichgewichtsregulation durch cerebelläre und kortikale Musterverarbeitungsprozesse und Speicherinhalte. Gleichgewichtsfunktionsstörungen können an den verschiedensten Orten der genannten Rezeptoren, wie auch innerhalb des Zentralnervensystemes entstehen. Für die diagnostische Suche [...]]]></description>
			<content:encoded><![CDATA[<p>Die menschliche Gleichgewichtsfunktion wird durch ein sogenanntes Raumkonzept mit Einspeisung von Informationen aus der Gleichgewichtstetrade, nämlich Auge, Vestibularis, Cochlea und Propriozept im menschlichen Hirnstamm gebildet. Ergänzt wird die Gleichgewichtsregulation durch cerebelläre und kortikale Musterverarbeitungsprozesse und Speicherinhalte.</p>
<p>Gleichgewichtsfunktionsstörungen können an den verschiedensten Orten der genannten Rezeptoren, wie auch innerhalb des Zentralnervensystemes entstehen. Für die diagnostische Suche nach den Läsionsorten, wie auch nach der Art der Störung bedient man sich verschiedener Kopfsinnesbahnen, die objektiv und quantitativ vermessen werden. Die klassischen objektiven und quantitativen äquilibriometrischen Verfahren sind die der sensomotorischen Augenbewegungskontrolle mittels Elektronystagmographie bzw. die der motorischen Kopf-Körper-Bewegungskontrolle mittels der Cranio-Corpo-Graphie u.a.m.. Für die synoptische Auswertung des Bárány&#8217;schen Kalorisationsversuches benutzen wir das Schmetterlingskalorigramm. Für die Steh- und Tretversuchanalyse bedienen wir uns der radarbildähnlichen Cranio-Corpo-Gramme. Neu hinzugetreten ist die kortikale Reaktionsauswertung mittels Brain Electrical Activity Mapping (BEAM) und vestibulär evozierter Hirnpotentiale (VbEP).</p>
<p>Alle genannten Verfahren werden miteinander vernetzt angewendet. Sie dienen nicht nur der Diagnostik, sondern auch der Steuerung einer modernen Differentialtherapie von Vertigo- und Nauseazuständen.</p>
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		</item>
		<item>
		<title>Craneocorpography test review</title>
		<link>http://www.neurootology.org/archives/296</link>
		<comments>http://www.neurootology.org/archives/296#comments</comments>
		<pubDate>Sun, 01 Jan 2006 00:00:00 +0000</pubDate>
		<dc:creator>admin</dc:creator>
				<category><![CDATA[Diseases]]></category>
		<category><![CDATA[Neurootology]]></category>
		<category><![CDATA[Propioceptive]]></category>
		<category><![CDATA[Propioceptive tests]]></category>
		<category><![CDATA[Craniocorpography]]></category>
		<category><![CDATA[Craniocorpography stepping test]]></category>
		<category><![CDATA[Vertigo]]></category>

		<guid isPermaLink="false">http://www.neurootology.org/?p=296</guid>
		<description><![CDATA[Many authors have reported different useful studies on the evaluation of the nystagmus which may be spontaneous and induced, any of these authors have been devoted to the study of vestibulo-spinal reflexes.1,2 The diagnosis of patients in modern neuro-otological centres has to include not only the history and examination, but also objective and quantitative tests. [...]]]></description>
			<content:encoded><![CDATA[<p>Many authors have reported different useful studies on the evaluation of the nystagmus which may be spontaneous and induced, any of these authors have been devoted to the study of vestibulo-spinal reflexes.1,2  The diagnosis of patients in modern neuro-otological centres has to include not only the history and examination, but also objective and quantitative tests. 3,4,5<br />
In this study should be pointed out the importance of a short but significant vestibulospinal equilibrium test, which is recorded by Claussen Craniocorpography (CCG).  The stepping test was first described by Unterberger, Fukuda, Peiterson and Zilstorff-Pederse in 1963.  In 1978, Claussen described the photographic technique for recording the stepping test, which was called CCG.6,7,8,9<br />
The vestibular functions can be tested involving two major systems:<br />
-Vestibulo-spinal = CCG<br />
-Vestibular-ocular= Nystagmus (ENG)</p>
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		<title>Approccio diagnostico ai disturbi dell’equilibrio negli esiti di colpo di frusta</title>
		<link>http://www.neurootology.org/archives/300</link>
		<comments>http://www.neurootology.org/archives/300#comments</comments>
		<pubDate>Sun, 01 Jan 2006 00:00:00 +0000</pubDate>
		<dc:creator>admin</dc:creator>
				<category><![CDATA[Diseases]]></category>
		<category><![CDATA[Neurootology]]></category>
		<category><![CDATA[Propioceptive]]></category>
		<category><![CDATA[Propioceptive tests]]></category>
		<category><![CDATA[Craniocorpography]]></category>
		<category><![CDATA[Vertigo]]></category>
		<category><![CDATA[Whiplash]]></category>
		<category><![CDATA[Whiplash Injury]]></category>

		<guid isPermaLink="false">http://www.neurootology.org/?p=300</guid>
		<description><![CDATA[The goal of this work is to study the vestibular system of whiplash people using a 3D dCCG system. A new experimental set-up, based on optoelettronic technique, was developed to evaluate the 3D pattern of movement of head and trunk in different experimental set-ups. The exam protocol is very close to the classical one with [...]]]></description>
			<content:encoded><![CDATA[<p>The goal of this work is to study the vestibular system of whiplash people using a 3D dCCG system. A new experimental set-up, based on optoelettronic technique, was developed to evaluate the 3D pattern of movement of head and trunk in different experimental set-ups. The exam protocol is very close to the classical one with some changes. The frequency of the steps is decided by the subject and not pre-arranged. Moreover the stepping tests is changed too: in dCCG there is a complete deprivation of sensorial afferents while in this case the parameters are evaluated in different conditions such as the complete deprivation of the sensorial afferents, the deprivation of some sensorial afferents or the presence of all the sensorial afferents. Furthermore the subject must take his hands and arms behind the back.</p>
<p>The displacement and rotation of the two rigid bodies head and trunk were analyzed to introduce new parameters with respect to the 2D dCCG. So 6 markers were adopted: three were put on an helmet placed on the head, two on the left shoulder and one on the right shoulder.</p>
<p>Subjects were asked to step on the spot in a more natural way and to standardize the length of the tasks, the test finished after almost 60 steps and 35 seconds.</p>
<p>Whiplash people executed four tests: open eyes, closed eyes, with and without the carpet on the floor. The considered parameters were extrapolated and generalized starting from those generally obtained by the 2D analysis, but trying to assess more the behaviour of the head and trunk (thought as two rigid bodies), than considering the trajectory of each marker. In particular the Index of Coordination, speeds (global with its lateral, vertical and longitudinal components), rotation angles (in the frontal, horizontal and sagittal plane) and displacements of the head and trunk were analyzed and compared with the normalcy databases.</p>
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